Mycoplasma pneumonia refers to an acute upper respiratory tract infection disease caused by Mycoplasma pneumoniae infection, which often includes pharyngitis, bronchitis and pneumonia at the same time.
Mycoplasma pneumonia is spread through the air through oral and nasal secretions and is distributed throughout the year, so it can cause minor epidemics of respiratory tract infections.
Mainly seen in children and adolescents, but also more common in adults. Although children aged five and above are the main high-risk group, immunocompromised people and special groups also need to be highly concerned.
NMN is also called β-Nicotinamide Mononucleotide. NMN is a substance inherent in the human body and constantly updated. NMN is the precursor of NAD+, and its function is reflected by NAD+. NAD+ is not only a key communication factor between the nucleus and mitochondria, but also an indispensable and important raw material for the human body's DNA self-repair system.
Inflammation is one of the leading causes of lung disease. Prolonged inflammatory responses may lead to airway remodeling and fibrosis, ultimately leading to decreased lung function. NMN can reduce inflammatory damage by inhibiting the inflammatory response. Studies have shown that NMN can inhibit the NF-κB signaling pathway, thereby reducing the production of inflammatory factors.
At the same time, NMN can also increase the activity of SIRT1 protein and further inhibit the inflammatory response. In addition, NMN can also reduce the concentration of inflammatory cytokines such as IL-6 and TNF-α, thereby reducing the degree of inflammatory response.
Oxidative stress is also one of the important factors leading to lung diseases. When oxidative stress is excessive, it can cause damage to cell structures, cause inflammation and reduce lung function. NMN has significant antioxidant effects and can effectively scavenge excess free radicals. Studies have shown that NMN can enhance the antioxidant capacity of cells by activating the SIRT1 pathway and increasing the activity of antioxidant enzymes (such as SOD, CAT and GPx).
In addition, NMN can also activate the Nrf2 pathway and promote cells to produce antioxidant factors. These antioxidant mechanisms make NMN a potential drug for the treatment of lung diseases related to oxidative stress.
Lung health cannot be maintained without the support of the immune system. NMN is a substance that has the ability to regulate immune responses and can affect the function of immune cells. Studies have shown that NMN can improve the phagocytic capacity of macrophages, thereby more effectively clearing infection and inflammatory factors.