Product News

NMN (nicotinamide mononucleotide) is the product of nicotinamide phosphoribosyl transferase reaction and is one of the key precursors of NAD+.

Study shows that nicotinamide mononucleotide restores the fertilization ability of oocytes exposed to environmental pollutants

The addition of glutathione can have an unexpected effect: it can be added to noodle products to have a reducing effect.

Combined with vitamin C: In addition, L-Glutathione Reduced vitamin C is also an important antioxidant in the body.

Glutathione has a broad-spectrum detoxification effect.

NMN and metformin bound to microscopic nanocrystals called silver quantum dots enhance metabolic function using substantially lower doses in mice.

Glutathione can not only eliminate free radicals in the human body, but also improve human immunity. Glutathione maintains health

Glutathione is widely present in animals and plants, L-Glutathione Reduced and plays an important role in organisms.

The abbreviation for glutathione, L-Glutathione Reduced and there is a noun to explain it as Melitin GSH. Melitin GSH is a special substance for detoxification.

A research duo from WashU and MIT discusses the progress in determining cell enzyme consumers of NAD+ and their link to the anti-aging therapeutic NMN.

This combination treatment is a promising strategy to attenuate damage in aged hearts

One week of nicotinamide riboside (NR) supplementation did not affect whole-body metabolic adaptation before, during, or after working out

Nicotinamide mononucleotide represents an effective protective mechanism to maintain healthy endothelial cells

Study shows that NAD+ boosting reduces age-linked amyloid deposition and restores mitochondrial balance in muscle cells across species

Research supports NMN as a potential method to combat the intergenerational consequences of the obesity epidemic

Acipimox enhances human skeletal muscle mitochondrial function in clinical study

SBI-797812 turns NAMPT into a “super catalyst” that more efficiently generates NMN

Age-associated mitochondrial DNA point mutations reduce oocyte quality in humans, providing a potential biomarker for embryo viability in assisted reproduction